Fats a.k.a. Lipids - and the Centenarian Diet
As it now turns out Saturated Fatty Acids (SFAs) are good, in fact essential. They help reduce insulin resistance and type 2 diabetes, (1). Present findings support adverse metabolic effects of refined carbohydrates and sugars and could also implicate other diseases, when there is a lack of SFAs. (2) The war on SFAs and Cholesterol is over and the evidence is being reviewed and conclusions reversed. (3,4,4a) Cholesterol is a health-promoting substance.
The body can make all the
Cholesterol it needs, dietary cholesterol has no effect on cholesterol blood levels, but dietary source are better as they are more
efficient within the body. (4b,4c) Ref: ... Cholesterol - Your Life Depends on It, Dr. Chris Masterjohn.
The two most important things we can do is to eliminate excess Carbohydrates and bring our Fatty Acid (FA) consumption back to a ratio more near our ancestors. Today, most people are eating too many Omega-6 fatty acids and this distorts the Omega-6 to Omega-3 ratio. This distortion of polyunsaturated fatty acids Omega-6 PUFAs, by Chris Dresser (Update) is a factor in the western diet that is causing inflammatory diseases.
The disastrous "low-fat diet" dogma of the last half century has led to a devastating drop in most people's intake of healthy saturated fats, including MCTs, as most people have bought into the erroneous assumption that saturated fats are unhealthy and will raise their risk of heart disease. (Note 1,2)
In a Nutshell | Avoid Vegetable Oils High in Omega-6
The single most important thing you can do to reduce your Omega-6 intake is to avoid processed seed and vegetable oils high in Omega-6, as well as the processed foods that contain them.
The above chart - Dietary Fats - shows some common fats and oils. Avoid all that have a high proportion of Omega-6 (blue bars). Also, an excellent presentation Good Fats, Bad Fats, at Optimizing Nutrition, by Marty Kendall, as well as Chris Dresser's - An Update on Omega-6 PUFAs - 2016. (Update)
foods/fats were only introduced to humans in the past 100 years and they have completely distorted the natural balance of these essential fatty acids. (Note 2)
A direct link with
Corn Oil and obesity and type II diabetes has been established (Inflammation diseases). Epigenetics is establishing the mechanisms of gene expression and diet.
Nutrigenomics, Epigenetics, and Stress Tolerance, Dr. Rhonda Patrick | april/15 (Patrick 1,2)
Once Upon a Time our farm animals were just as healthy as
Fish, Ref: ...
D. S. McGerk - Where did the Omega 3's Go - Fat and Oil in modern diets | contributor April/2016
Dr. Cate Shanahan provides a more complete chart of the fats and oils and how they should be used in our diet. Dr. Cate breaks down and list fats as
Ok, but Not Great, and
These are categorized and include a simply
Dr. Cate Shanahan at Low Carb Down Under.
Practical Lipid Management for LCHF Patients - Jul/2016
Dr. Cate - WHY VEGETABLE OILS ARE TOXIC and she explains in the video and summarizes ... these oils contain a high percentage of polyunsaturated fatty acids (PUFAs). PUFAs are unstable, and break down rapidly when exposed to chemical stress. The refining process exposes these PUFAs to heat, pressure, metals and bleaching agents, and chemically alters the molecules into a wide variety of potent toxins with long names like 4-hydroxynonanal and 4-hydroxyhexanol, aldehydes, and others. These molecules are toxic because they promote free-radical reactions that damage our cellular machinery including mitochondria, enzymes, hormone receptors, and DNA. (Note - Dr. Cate)
Omega-3 Balance is Important!
Siscovick, DS, et al. Dietary intake and cell membrane levels of long-chain n-3 polyunsaturated fatty acids (LCPUFAs) and the risk of primary cardiac arrest. (5)
Omega-3 Index: is a test that can be performed and will shed light on the risk factors for death from coronary heart disease. Omega-3 fatty acids, also called n-3 polyunsaturated fatty acids (n-3 PUFAs), are involved in multiple biological pathways, including coagulation, muscle function, cellular transport, and cell division and growth. Low intakes or blood levels of eicosapentaenoic and docosahexaenoic acids (EPA + DHA) are independently associated with increased risk of death from coronary heart disease (CHD).
There are two essential fatty acids (EFAs) in human nutrition: α-linolenic acid (αLA), an omega-3 fatty acid; and linoleic acid (LA), an omega-6 fatty acid. Thesa omega-3 and omega-6 fatty acids cannot be produced by the human body. These fatty acids must come from food and are therefore called essential fatty acids - EFAs, (Wiki). We just do not need the overload of omega-6s we are getting in the SAD.
We cannot make an intelligent decision of Red Blood Cell Fatty Acids without analyzing “All” fatty acids,
- saturated fatty acids,
- monounsaturated fatty acids,
- omega-3 polyunsaturated fatty acids (n-3),
- omega-6 polyunsaturated fatty acids (n-6),
- and trans fats.
The Five main categories of fatty acids that can be found in red blood cells;
The last 3 are currently
Completely ignored by Medicine's SOC. This is principally because of the belief in the “Cholesterol Theory”, which has NO scientific basis,
and has Failed over the Last 40 years. The battle against cholesterol and saturated fats has Produced No Positive Results, in fact, it can clearly be shown to have produced
Type 2 diabetes, Insulin Resistance = Metabolic Syndrome, and the modern diseases of Inflammation High Carbohydrates - was not the answer.
The Omega-3 Index reflects the relative amount of EPA + DHA in red blood cells. It is expressed as the percentage of the total amount of fatty acids present. In fact it’s quite simple; if 8% of all the fatty acids present in red cell membranes is EPA+DHA, the Omega-3 Index is 8%. (Note 3,4,5)
In randomized secondary prevention trials, fish or fish oil have been demonstrated to reduce total and CHD mortality at intakes of about 1 g/day. Red blood cell (RBC) fatty acid (FA) composition reflects long-term intake of EPA + DHA. (Note 3)
- Example: farm raised fish (soy pellets as food), die of the same diseases as CAFO farm animals ... CHD - heart failure. Ref: The Critical Differences Between Omega-3 Fats From Plants and Marine Animals | Mercola, september, 2016.
Why Care About | Omega-6 - Omega-3 Ratio?
Our ratio of Omega 3 to Omega 6 fats over the last 100 years has changed for the worst. We eat way too many Omega-6 fatty acids while at the same time animal foods Omega-3
fat content has dropped due to feedlot and factory farming practices (CAFO - Confined Animal Feeding Operations)
and is now the lowest it has ever been. (Note 2) Ref:
The rise of Omega 6 fats, and where did the Omega 3's go from our food sources
... D. S. McGerk | contributor, may/16
Siscovick, DS, et al. Dietary intake and cell membrane levels of long-chain n-3 polyunsaturated fatty acids (LCPUFAs) and the risk of primary cardiac arrest. (5)
A distorted ratio of these polyunsaturated fatty acids may be one of the most damaging aspects of the Western diet. In contrast to omega-3 fatty acids, omega-6 fatty acids (eg, arachidonic acid [AA]) and their metabolites are more pro-inflammatory than anti-inflammatory. Anthropological evidence also suggests that the ratio human beings evolved eating is somewhere around 1:1, while the ratio today is about 16:1. (6,6a)
Excessive amounts of omega-6 polyunsaturated fatty acids (PUFA) and a very high omega-6/omega-3 ratio, as is found in today's Standard Western Diet (SAD), promote the pathogenesis of many diseases, [Simopoulos, 2002] (6,6a) including cardiovascular disease, cancer, and inflammatory and autoimmune diseases, whereas increased levels of omega-3 PUFA (a low omega-6/omega-3 ratio) exert suppressive effects. In the secondary prevention of cardiovascular disease, a ratio of 4/1 was associated with a 70% decrease in total mortality.
- Number one, don't eat vegetable oils high in n-6 fats.
- Number two, find a source of n-3 fats that can approximately balance your n-6 intake.
|Omega-6 Fatty Acids||Omega-3 Fatty Acids|
|Linoleic acid||LA||18:2n-6||Î±-Linolenic acid||ALA||18:3n-3|
|Î³-Linolenic acid||GLA||18:3n-6||Stearadonic acid||SDA||18:4n-3|
|Dihomo-Î³-linolenic acid||DGLA||20:3n-6||Eicosatetraienoic acid||ETA||20:4n-3|
|Arachidonic acid||AA||20:4n-6||Eicosapentaenoic acid||EPA||20:5n-3|
|Adrenic acid||22:4n-6||Docosapentaenoic acid||DPA (n-3)||22:5n-3|
|Tetracosatetraenoic acid||24:4n-6||Tetracosapentaenoic acid||24:5n-3|
|Tetracosapentaienoic acid||24:5n-6||Tetracosahexaenoic acid||24:6n-3|
|Docosapentaenoic acid||DPA (n-6)||22:5n-6||Docosahexaenoic acid||DHA||22:6n-3|
If we want
Live to 100 years of age, we have to rethink our eating habits. From the Fatty Acid Chart, we see why the Mediterranean diet has gained such prominence. Olive Oil is the best
under this criteria. Even grass-fed butter can is best, we get over 400 nutrients and fats from butter. Omega's 6:3 Ratio | contributor Apr/2016
Flaxseed oil seems to be best until you look at the chemical formula. Humans require C20:5n-3, and C22:6n-3, EPA and DHA. Flaxseed oil is ALA C18:3n-3, which our bodies are poor at converting to usable forms. It is much better to use the
However, studies of LA vs CLA (C18:2 c-9, c-11, from ruminant animals) (9) showed dramatic decreases in cancer incidence with CLA. In mice, 80% of those on a normal diet got breast cancer while only 40% of those consuming CLA were found to have cancer. A 50% reduction in cancer incidence! (10,10a)
Cancer metastases occurred in 80-100% of mice fed the linoleic acid versus only 10% of CLA-fed mice. Once again, a very dramatic difference. (11) Therefore, for this reason, animal sources of Omega-3 from fish and grass-fed ruminant animals (fermenting stomach) are best for human use ... D. S. McGerk - Where did the Omega 3's Go? | Contributor, april/16
A specific beneficial fatty acid called conjugated linoleic acid (CLA) is associated with weight loss, according to the May 2007 American Journal of Clinical Nutrition [Whigham, L D, et al. 2007]. (11a) The content of conjugated linoleic acid (CLA) in dairy products increases as a function of pasture in the cow's diet.
This indicates the importance of healthy fatty acids in the body's Metabolism of Fats as CLA is indicated in weight control. (11b) Ref: ... Cheesy Charlie - Cheese - What is Best for Health? | Contributor, jun/16
- Modern oils are a big fat - NO = n6 PUFAs. Converting a vegetable or plant-based food into oil is a complicated high heat chemical process, as the oil is not simply
squeezed out of the vegetable, with the exception of
Cold Pressed Oils. Intense chemical manipulation is required, Reference: Dr. Colin Champ, "Caveman Doctor" Vegetable Oils: The Refining of Our Health. (12,12a)
What's left, Boils down to, Avocado Oil and Ghee (butter - clarified, i.e. no milk solids or water), (High heat), Olive oil, Coconut Oil, and Grass fed butter, (medium heat), Olive and Avocado oils, (Salads) i.e. cold pressed oils
and non-processed fats. Although, there is no evidence that 200,000 years ago we cooked with oils. More than likely everything was
Fire Roastedas the earliest known pottery is dated to about 20,000 years ago, thusly, cooking on sticks (marshmallow style) was probably the norm. (13)
From modern research we know that high temperature cooking creates Trans-Fats
(14) which are undesirable. Therefore saturated fats are best for high temperature cooking simply because there are no double bonds in the carbon to carbon bond to break and oxidize.
Saturated fats have no unexposed weak points for the by-products of metabolism i.e. free-radicals to attack: (12,12a)
Versus: PUFAs (poly = many) have many sites that can be attacked by free radicals: (12,12a)
From: An Investigation into the Association between DNA Damage and Dietary Fatty Acid in Men with Prostate Cancer, Nutrients 2015, Karen Bishop, et al. (15)
The biosynthesis pathways of the omega 3, 6 and 9 family of poly- and mono-unsaturated fatty acids.The main dietary sources are shown in blue text, and the enzymes in red text. EPA and DHA from fish oils show favorable effects in inflammatory bowel disease, some cancers and cardiovascular complications. (16,16a,b,)
Cholesterol, Lipoproteins and the Liver:
Cholesterol is essential to life; it is a primary component of the membrane that surrounds each cell and functions as a precursor to fat-soluble vitamins and steroid hormones.
Cholesterol is not technically a "Fat"; rather, it’s classified as a Sterol (Wiki), which is a combination of a steroid and alcohol. Cholesterol is vital to animal cell membrane structure and function as a precursor to fat-soluble vitamins and steroid hormones. In order for cholesterol to be transported around the body in the blood, it is carried by special proteins called lipoproteins. These lipoproteins are classified according to their density; two of the most important in cardiovascular disease are low-density lipoprotein (LDL) and high-density lipoprotein (HDL).
Nina Teicholz - The Big Fat Surprise explains at TEDxEast why everything we thought we knew about dietary fats is wrong. Dietary sources of cholesterol are more efficient to the body. It is a 25 step process for the liver to make cholesterol. Dr. Stephanie Seneff explains the Cholesterol and Vitamin D connection within your skin.
Cholesterol is so important that our bodies can make all it needs and can make cholesterol out of ANYTHING - fats, carbohydrates, or proteins. Since every single animal cell contains cholesterol, all animal foods contain cholesterol. Many people don’t realize that all muscle meats (chicken, fish, beef, pork, etc.) contain about the same amount of cholesterol per serving.
Cholesterol and Saturated Fats - De-bunked: the greatest mistake made by
Government - is Bad Science or
Distorted Data. Governments are responsible to its citizens and for 40 years of
Anti-Saturated Fat the governmnet has been wrong and have allowed
Special Interests to demonize fats. Explaining how fats became a dirty word is exposed by
David Diamond, PhD - Demonization and Deception in Cholesterol Research | September/2015., and if you
take the time to write down and reseach all the science in this video you will be well informed.
The liver is central to the regulation of cholesterol levels in the body. Not only does it synthesize cholesterol for export to other cells, but it also removes cholesterol from the body by converting it to bile salts and putting it into the bile where it can be eliminated in the feces. (20)
Cholesterol, Lipoproteins and the Liver, University of Washington (20,20a)
An open discussion on this mechanism is presented by Dr. Rhonda Patrick's PodCast with Dr. Ronald Krauss on LDL Cholesterol | october/15 (Video), and the conclusion that simple sugars, particularly fructose are the cause of the imbalance in blood lipids. A low fat diet promote small LDL particles which are more dangerous indicating that LDL is not a good indicator. Low fat intake coupled with a high carbohydrate, which is the SAD, promotes bad nutrition.
Dr. Fettke also has supporting evidence on his web site NoFructose.com where is states
Inflammation is at the heart of all disease and our modern diet is to blame. Inflammation is caused by the current excessive consumption of the combination of Fructose, refined Carbohydrate and Polyunsaturated oils.
The last 10 years and current research will put an end to
The Saturated Fat - Cholesterol Theory
a theory that has
No Scientific Evidence to support it. In 1961 a relational study, (Ancel B. Keys) (22), was done on cholesterol, his observations changed the political will of the AMA, AHA and
Washington Politicians. Political powers were convinced that saturated fats and cholesterol were bad, while the real culprit the
Refined-Carbohydrate Disease (23), was ignored. Ref: ... the Obesogenic Diet.
Our Fav - Ivor Cummings ...
it is Not Cholesterol but Insulin as the Major cause of Heart Diseases (CAC) - Jan/2017
Your body makes all the cholesterol is needs and “Cholesterol is vital to body Metabolism”. (24) (Lipoprotein metabolism) Eating cholesterol has no effect on this process, consequently, if your blood pressure, triglycerides, and cholesterol are out of whack - "Metabolic Syndrome" ... your diet and lifestyle are screwed up ... Your fault!
And Ref: our own contributors ... D. S. McGerk - Carbohydrate Intolerance - What does this mean? | contributor feb/2017 ... Marilyn's Column - Carbohydrates and Longevity living to 100 | contributor Oct/2016 ... D. S. McGerk - Fat and Oil in modern Diets, Where did the Omega-3s Go | contributor Apr/2016.
- If you want to enjoy healthy fats with a lot of flavor, then you cannot have simple sugars. ... this site, live to 100 do not eat foods. You cannot have both, and of course refined carbohydrates have never been part of the human diet prior to the 20th century.
The warnings about Sugar, White flour, and Starchy vegetables started in the 18th century (150 years ago) with Dr. Tanner -
The Practice of Medicine, 1869 (25) and a book by William Banting on LCHF eating
Letter on Corpulence, 1864 (25a). An american doctor, Dr. Emmet Densmore also wrote a medical book
How Nature Cures - The Natural Food of Man, 1892 (25b) and on the cover his mission states ...
A statement of the principal arguments against the use of bread, cereals, pulses, potatoes, and other starchy foods.
Hypothesis: there is evidence that
Cholesterol deficiency coupled with not enough actual sun and hence a vitamin D deficiency,
and with a deficiency in sulfur, defeats the purpose of cholesterol, and Cholesterol-Sulfate as a metabolic agent and an energy source for the body.
The body will pull/steal sulfur from the
Myelin sheath and
Collagen in the Joints. Is this "Triage" at work leading to neuropathy and arthritis.
Dr. Stephanie Seneff explains that if there is not enough cholesterol-sulfate in the blood your cell walls can be damages and present as dysfunction. All cells require cholesterol to function. Discussion on sulfur: Dr. Mercola Interviews Dr. Stephanie Seneff (Part 3 of 7) (29,29a)
She points out that heart disease may actually be related to cholesterol sulfate deficiency, and explains how elevated LDL cholesterol is a sign of this deficiency. In short, high LDL (incorrectly referred to as “bad” cholesterol) is your body’s way of compensating for cholesterol sulfate deficiency. This sounds a little like Dr. Ames' - Triage Theory at work ...
"Heart disease, I think, is a cholesterol deficiency problem, and in particular a cholesterol sulfate deficiency problem..." says Dr. Seneff.
The ongoing Framingham Study (Wiki) found that there was virtually no difference in coronary heart disease "events" for individuals with cholesterol levels between 205 mg/dL and 294 mg/dL - the vast majority of the US population. Even for those with extremely high cholesterol levels - up to almost 1200 mg/dL, the difference in CHD events compared to those in the normal range was trivial. (29b)
Very Anti-Statin ... essentially, by elevating LDL, your body is protecting itself from the harmful effects of cholesterol sulfate deficiency. When you simply remove the LDL (medicate), you remove this "backup" mechanism aimed at keeping your heart going strong. As a result, heart failure becomes a distinct possibility. Just one of "hundreds" of known and possible side effects of "Statins" i.e. Mayo Clinic ... Statin side effects: Weigh the benefits and risks.
Dr. Georgia Ede - Diagnosis: Diet points out that “Statin” drugs, interfere with the insulin function which tells the cell to make more cholesterol. If your cells need more cholesterol due to growth and cell replacement, insulin turns the enzyme HMG-CoA reductase ON, but when a statin drug is blocking the critical enzymes in this pathway, your cells may not be able to make cholesterol when needed. And what’s worse is that the cholesterol synthesis pathway doesn’t just make cholesterol; but, branches of this same pathway are responsible for synthesizing a wide variety of other important molecules, including: Vitamin A, Vitamin E, Vitamin K, and Coenzyme Q. (Note 4)
Hypothesis: ... we would not be able to move if our cell wall were not composed of Omega 3's, good PUFA's, and cholesterol. We are 70% water, and remain flexible.
Fish on the other hand, can swim in 32° water and remain flexible. How is that possible? If that was aCowor aHuman, we would be a solid block (i.e.Stiff as a Board).
Hypothesis Proof | The Body's cellular integrity
n6 PUFAs have many double bonds and consequently remain
Flexible. Without the double bonds the fats would be solid and non-flexible.
In addition, we know that
High Fat content displaces water as lipids are hydrophobic - "water-fearing" (Wiki) and are higher
density 9kcal/g, whereas both glycogen and proteins are hydrophilic - "water-attracting" (Wiki) and we would need to be physically much larger
because 1-1.5kcal/g "wet weight" would require 6 times more cellular space.
Fish can regulate their body water content to as low as 30%. This give them a huge advantage in
Cold Water and very beneficial as a food source by providing essential Omega 3 lipids.
TBD ... more ...
Conclusion: this is the main reason that our cell walls are made out of Proteins and Lipids. Cholesterol is a critical building block of our cells and works in conjunction with the skin and Vitamin D. Dr. Stephanie Seneff has shown the connection of Cholesterol - Sulfur - Vitamin D. (28,28a,b)
TBD ... more ...
Adapted from Das UN. Biotechnol J. 2006 Apr;1(4):420-39
According to Chris Dresser Vegetarians and Vegans Should Supplement with DHA. His article ... dated September, 2016 gives a good explanation of DHA and EPA and discusses the differences between Omnivores, Vegetarians, and Vegans.
We discuss this above "Why Care About Omega-6 and Omega-3 Fatty Acids" and it is pointed out that ALA conversion gets interfered with due to interactions of PUFAs, and the comversion rate to DHA and EPA are less that 1%.
Ketosis is not Ketoacidosis
Ketosis ≠ Ketoacidosis - Ketones are Fuel
Ketoacidosis (aka Diabetic Ketosis - DKA) is a life-threatening condition in which your body doesn’t make enough insulin. This can cause you to have dangerously high levels of ketones and blood sugar. This combination makes your blood too acidic, which can change the normal functioning of internal organs like your liver and kidneys. If you don’t get prompt treatment for it, you could go into a coma or die.
This was first discovered in the Urine of Type I diabetics. A high level of Ketones (Wiki) were measured in the urine indicating that the body was not processing carbohydrates. Poor diabetes management is a leading trigger for DKA. But today, we know that Type II diabetes can be over come or controlled with a LCHF diet. Avoiding MetS - Metabolic Syndrome is a key issure with lifestyle. Ref: Addictions ... the Obesogenic Diet.
Ketosis is a normal state of metabolism, whereby, our bodies move in and out of Fat Burning mode. If you have ketosis, you have a higher than usual level of ketones in your blood or urine. Ketones are a chemical your body produces when it burns stored fat.
Ketones are a more efficient energy fuel for our metabolism. A low carbohydrate diet can trigger ketosis. That is because a low-carb diet will cause you to have less glucose in your blood, which will, in turn, cause you to produce less insulin. Your body will begin to burn fat for energy instead of relying on sugars. Ref: Carbohydrates and Longevity | contributor october/2016.
The Pyramid indicates that in both cases of either
Ketosis or Ketoacidosis the body's first line of defense is
Low Carbohydrates. Low carbs promote the body's optimum metabolism and an example of this
was published in 1996 by S. G. Hasselbalch, et.al., in the American Journal of Physiology ...
A ketogenic state results in a substantial (39%) increase in cerebral blood flow, and appears to reduce cognitive dysfunction associated with systemic hypoglycemia in normal humans.
Also on this site ... Carbohydrate Intake - Prevents Fat Burning
Best selling author Nora Gedgaudas - Primal Body Primal Mind goes beyond the Paleo Diet for total health and a longer life, (i.e. becoming a Centenarian). She has good a video back by hard science, and has her own popular podcasts are on iTunes ... or her site ... Primal mind: Nutrition and mental health - Improving the way you feel and function, cultivating an ageless mind, by Nora Gedgaudas. (32,33)
Ok - Now What?
Our modern society created a Nutritional Disease; therefore, we need a Nutritional Solution. But, doctors are not trained in nutrition, medicine as we know
it is in trouble, can we be better doctors than the AMA provides. We certainly can acquire the knowledge to ask better questions. The AMA has failed in their mission. Dr. Noakes gives a good talk on the problem ...
The Great Diet Controversy: UCT - Challenge Beliefs | Dr. Tim Noakes, november/2013
(30) and Dr. Mercola & Dr. Greger on How Not To Die | December/2015.
Dr. Mercola and Dr. Chutkan talk on
Food as Medicine - The Baby Connection and the importance of Gut Health ... referred to as
Complimentary or Alternative medicine ...
Dr. Mercola and Dr. Chutkan on Gut Health | February/2017. (32)
Dr. Chutkan has a 77% success rate of taking her patients
The current issue is that everyone responds differently to Carbohydrate Sensitivity → Intolerance. Hence, we see people that can use Sugars and many who develop chronic inflammation and autoimmune diseases. Ref: ... Insulin Resistance.
Exercise ... is Good Stress, so avoid a sedentary lifestyle and avoid the
Colloquial Tag - Couch Potato.
The body has three to six (3-6) principle circulatory systems, depending on definitions and
All require muscle movement to function.
→ Cardiovascular - Heart, → Pulmonary - Lungs,
→ Systemic -Blood and Vessels, moved by our involuntary muscle system.
Simple solution, avoid carbohydrates as they offer no known nutritional benefits. Carbohydrates are however packaged in food source organisms that do provide Longevity Vitamins. We want and need Phytochemicals from our Diet, otherwise we suffer from malnutrition. The symptoms of malnutrition is what this site is all about.
Our foods provide Phytonutrients, macronutrients, micronutrients (for critical DNA repair) (R1), Polyphenols, Phytosterols, Flavonoids, Lignans, Antioxidants, and Carotenoids, that do allow life to continue. Ref: 100 richest dietary sources of polyphenols. (Resource 1,2,3)
Certainly avoid processed carbohydrates, fats and oils, and packaged and processed foods. A direct result of too many carbohydrates is Insulin Resistance functional manifested as Carbohydrate Intolerance. Dr. Volek discusses his current views on the benefits of a LCHF plan - Health-Promoting Effects of a Low-Carbohydrate Lifestyle | july/2016 and this site follows this type of plan with the addition of Dr. Ames' "Triage Theory" ... the Centenarian Diet.
- Modern oils are a big fat - NO = Omega-6 PUFAs. Converting a vegetable or plant-based food into oil is a complicated chemical process. Intense chemical manipulation is required. (35) The vitamin D made by your body last 3 times longer than supplement vitamin D, the reason is obvious in that there is much more going on with this manufacturing process. (36)
- Factory-farmed, or CAFO meat and animal products are loaded with omega-6 PUFA depicted here: Source: EatWild.com Therefore, grass-fed or New Zealand lamb and beef. Even Australia, like New Zealand, can be better, as they have unlimited range and little augmented feeding is required. 100 year ago, farm animals (including chicken) had the same nutrition as fish. Animal fats should be yellow "Not White", as the B vitamins are yellow as is the omega 3 fats. Our great grandparents used "Lard" and "Tallow", for cooking. Bacon fat was a favorite for frying eggs.
- Harris and Von Schacky propose that the RBC EPA + DHA (hereafter called the Omega-3 Index) be considered a new risk factor for death from CHD. Red blood cell (RBC) fatty acid (FA) composition reflects long-term intake of EPA + DHA. The Omega-3 Index was inversely associated with risk for CHD mortality. An Omega-3 Index of > or = 8% was associated with the greatest cardioprotection, whereas an index of < or = 4% was associated with the least. CONCLUSION: The Omega-3 Index may represent a novel, physiologically relevant, easily modified, independent, and graded risk factor for death from CHD that could have significant clinical utility. (37,38,38a)
- All living cells are constructed with cholesterol. Plants contain their own special forms of cholesterol called “phytosterols”, but phytosterols are toxic to human cells, so our intestines wisely refuse to absorb them.
- Most cholesterol from foods does not get absorbed unless body levels are low.
- The amount of cholesterol we eat has almost no effect on our cholesterol levels.
- The vast majority of cholesterol in our body is made by our body’s own cells. Any excess cholesterol is coming from our own body's manufacturing, not from the food we eat.
- The relationship of fish and dietary omega-3 fatty acids and cardiovascular disease (CVD) has been investigated in numerous studies and comprehensive reviews and recommendations exist, but guidance on blood concentrations is missing. Determination of blood omega-3 levels may help guide the appropriate use of dietary fish or omega-3 supplements in a personalized heart disease prevention strategy. (39,40,40a)